biochemistry
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biochemistry - Marcador
biochemistry - Detalles
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308 preguntas
| 🇬🇧 | 🇬🇧 |
| Increase absorption of nonheme iron | Hydrochloric acid |
| Decrease serum iron = decrease transferrin Saturation | Increase of iron binding capacity |
| Ferric = Ferrous via what enzyme | Duodenal cytochrome B |
| Enzyme that transport Iron to enterocyte | Heme carrier transporter 1 (HCP1) |
| Enzyme that separates Heme and Ferrous | Heme oxygenase 1 (HO1) |
| Heme contains what form of iron | Ferrous (Fe 2) |
| Non heme has a ______ type of iron | Ferric(Fe 3) |
| Ferric = Ferrous via what enzyme | Duodenal cytochrome B |
| DCYTB has an enzyme that reduces Fe3 to Fe2 ; | Ferric Reductase |
| Co-factor of non-heme transport | Vitamin C |
| Carries transformed non heme Fe2 into the enterocyte | Divalent metal transporter (DMT1) |
| _____is only absorbable compound of iron | Ferrous (Fe2) |
| Allows Fe2 to enter the circulation through this transport | Ferroportin transporter |
| Fe2 released by the Ferroportin must become a Fe3 due to | Fe2 cannot transported by the transferrin |
| What enzyme oxydizes Fe2 to Fe3 | Hephaestin |
| Hephaestin has an enzyme called | Ferrous oxidase |
| Increase copper | Increase in iron production |
| Binds ferric to transferrin | Iron transferrin bind complex |
| Reduce Fe 3 to Fe2 | Ferric Reductase |
| Channel absorbs nonheme Fe2 | Divalent metal transporter (DMT 1] |
| Reduced form of transferrin | Apotransferrin |
| Oxidize Fe2 to Fe 3 | Ferrous Oxidase |
| Iron transferrin complex binds to | Transferrin Receptor |
| Regulator of ferriportin | Hepcidin |
| Increase in hepcidin | Decrease of ferroportin |
| Stimulants hepcidin | Lipopolysaccharide |
| Stimulants hepcidin | Anti inflammatory mediator (IL6 ) |
| Stimulants hepcidin | Increase Fe 2 in blood |
| Stimulants hepcidin | Hemochromatosis Gene (HFE GENE) |
| Major regulator of hepcidin | HFE Gene ( hemochromatosis gene) |
| Mutation of HFE gene | No inhibition of ferroportin channel |
| Increase ferroportin | Increase iron in blood |
| Increase absorption of nonheme iron | MFP Factor |
| Promotes absorption of Fe3 | MFP FACTOR |
| Increase absorption of nonheme iron | Vitamin C |
| Increase absorption of nonheme iron | Lactic and citric acid |
| Increase absorption of nonheme iron | Hydrochloric acid |
| Inhibits iron absorption | Phylates and fibers |
| Inhibits iron absorption | Oxalate (spinach) |
| Inhibits iron absorption | Calcium and phosphorus |
| Inhibits iron absorption | Tannic acid |
| First indicator of iron deficiency | Serum ferritin |
| Decrease transport iron =_________ | Inactive hemoglobin transport |
| Genetic disorder caused by increase absorption of iron | Hemochromatosis |
| Chromosomal defect in hemochromatosis | Autosomal recessive disorder |
| HFE gene in hemochromatosis is located at | Chromosome 6 |
| Hereditary type of hemochromatosis | Hemochromatosis type 1 |
| Increase in this hormone will increase testosterone | Luteinizing hormone (LH) |
| Converts testosterone to estrogen | Aromatase enzyme |
| Increase in zinc will | Activate aromatase leading to decrease production of estrogen |
| Decrease zinc will | Increase estrogen due to inactivation of aromatase |
| Seen in alcoholic individuals | High estrogen, high aromatase, high fat deposit |
| Activates T-lymphocytes | Fe2 andZinc |
| Superoxide dismutase | Convert zinc, manganese, and copper to antioxidant |
| Glutathione peroxidase | Converts selenium to anti oxidant |
| Cofactors of Zinc | Peptidase, thymidine kinase, carbonic anhydrase, Lactate dehydrogenase alcohol dehydrogenase, alkaline dehydrogenase, superoxide dismutase |
| Relationship of Zinc to Prostate | Zinc activates citrate to decrease cell hypertrophy |
| Caspase cascade leads to | Apoptosis |
| Increase zinc = stimulation of mitochondrial pore | Release of cytochrome c |
| Cytochrome c activates; | Caspase cascade |
| BAX Gene activates | Mitochondrial pore to release Cyt C |
| Methallothionein | Regulate zinc released in blood |
| Binds zinc and copper | Methallothionine |
| Acrodermatitis Enterohepatica chromosomal defect | Autosomal recessive |
| Zinc and copper compete in | Binding to metallothionein |
| In Infection Fe decrease-while | Copper increase |
| Copper became antioxidant cause of whet enzyme | Superoxide dismutase |
| Stimulate secretion of CP from hepatocytes | Interlukin 1 and 6 |
| Synthesis and secretion of CP occur in the | Hepatocytes |
| Converts DOPA to DOPAMINE | L-DOPA Decarboxylase |
| Clinical manifestation of zinc deficiency | Acrodermatitis Enterohepatica |
| Zinc deficiency manifested in allergic reaction | Acrodermatitis enterohepatic |
| Increase in iron due to genetic increased absorption of Fe | Hemochromatosis |
| Large deposit of iron in tissue | Hemosiderosis |
| Enhances activity of different oxidases | Molybdenum |
| Oxidation of purines and pyrimidine producer of uric acid | Xanthine oxidase |
| Cooper ATPase enzyme | ATP7A and ATP7B |
| Manganese is a key component of all of the following enzymes | Argindse, pyruvate carboxylase , and Mn-superoxide dismutage |
| Goitrogenic foods are | Cassava and lettuce |