How is ANS role in HTN?

Adrenergic reflexes modulate BP over short term, NE, EPI and DA play a role, receptors divided into (a1,a2,b1,b2) a1 postsynaptic cell in smooth muscles and elicit vasoconstrictors, a2 activated by catecholamines negative controllers inhibit further NE release. b1 stimulates rate and strength of CO, and stimulates renin release from kidney, b2 EPI relaxes vascular smooth muscle results in vasodilation. Sympathetic outflow increase in HTN than normotensive.