What are hemodynamic pathophysiological mechanisms?

Hydrostatic and oncotic pressures guide movement of fluids, elevated osmotic pressure in venous circulation regulates increased hydrostatic pressure in arteries, forms small nte flow of fluid to interstitial space drained by lymphatic vessels, increased hydrostatic pressure or decreased osmotic increase water movement to interstitium, increases fabric pressure until reaches equilibrium again, excess edema fluid will be retained by lymph vessels. We may get increased hydrostatic pressure (obstruction of vein cause localized edema/global HF cause generalized edema), hydro-sodium retention (edema by increasing hydrostatic pressure and reducing osmotic pressure of plasma by excessive sodium retention seen in diseases compromising kidney function) Decrease osmotic pressure (reduced albumin, nephrotic syndrome (most imp cause of albumin loss), cirrhosis (albumin not secreted) cause secondary hyperaldo.) Obstacle in lymphatic drainage (localized obstruction by inflammatory/neoplastic [filariasis lower extremities/genitalia, infiltrates in breast, lymphedema due to radiation.