How is evolution of TB inflammation?

Exudative phase (congestion, edema, polynuclear influx, occurs only if we see large BKs number in Zeihl-Nelson stain, no suppuration but caseatous necrosis, which depends on immune status, yellow-white lesion eosinophilic anhist area) Tuberceloid granuloma (Around caseatous necrosis or in its absence in exudative phase, polynuclear cells disappear, lymphocytes and epitheliod cells which can merge form multinucleated cell, horseshoe arrangement (langerhans cells), grouped into nodules) Inflammation (not directly related to BK, more type IV HS, inhibit evolution to fibrosis by outbreaks of disease (new lesion) or presence of caseum in inflammatory focus) so we see lesions of different ages in chronic TB lung, can subsit for years)