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level: Drugs for Treating Hypertension

Questions and Answers List

Question	Answer
SNS agents centrally acting α2-agonist; causes decreased sympathetic outflow.	Clonidine
SNS Peripherally acting Uptake by transmittervesicles in nerve depletesand replaces norepinephrine in neurosecretory vesicles.	Guanethidine
SNS Peripherally acting α1-antagonist.	Prazocin
SNS Peripherally acting β-antagonist.	Propranolol
SNS centrally and Peripherally acting Binds tightly to storage vesicles, which consequently lose their ability to concentrate and store norepinephrine.	Reserpine
Arterial Vasodilators Opens K+ channels and causes hyperpolarization of smooth muscle.	Diazoxide
venous and Arterial Vasodilators Releases NO, which binds to guanylyl cyclase to generate cGMP.	Nitroprusside
Inhibits voltage-dependent “L-type” Ca++ channels	Nifedipine
Inhibits conversion of angiotensin I to angiotensin II.	Captopril
Inhibits Na+ channels in luminal membrane in the proximal segment of the distal tubule	Hydrochlorothiazide
Inhibits cotransporter of Na+,K+, Cl− in the ascending limb of the loop of Henle	Furosemide
Thiazides Diuretics	Hydrochlorothiazide
Ca++ channel-blockers	Nifedipine
Loop Diuretics	Furosemide
ACE inhibitors	Captopril