| life cycle of T solium | 1) eggs or gravid proglottis in feces( diagnostic/infective stage)
2) eggs/gravid proglottis eaten by pigs/human
3) oncospheres( infective) hatch, penetrate intestine and invade muscle. oncospheres develop into cystercerci in muscle of pigs and humans.
4) human ingest rew/ undercooked meat
5) scolex attach to intestine
6) adults in intestine |
| clinical feature of infection f T. solium | NEUROCYSTICERCOSIS: Seizures,hydrocephalus, arachnoditis, headaches, nausea, vomiting, visual impairment, papilloedema |
| diagnosis of T. solium include | CT. MRI and ELISA |
| treatment of T. solium infection | control seizure, praziquantol, albendazole. corticosteroids, antiparasitic drugs may exacerbate inflammation.
hydrocephalus, seizures |
| INfection caused by larva nematodes that migrate to tissue that cause intense eosiphillic respone? | Visceral& occular Larva migrans. most commonly caused by T. canis/ cati |
| clinical features of Visceral and ocular larva migrans | fever, malaise, anorexia, weight loss, cough, wheezing and rashes. |
| treatment of viscera and ocular larva migrans | infection is self limiting. use corticosteriods for inflammation and albenadiazole ofr larvae |
| cutaneous larva migrans | cuased by dog and cat hookworn ancylostoma braziliense. |
| treatment of cutaneous larva migrans? | treat with albendazole/ivermectin |
| the infective stage of T. solium | oncospheres, eggs or gravid proglottis |
| in human: cystercercosis may develop in any organ but more common in? | subcutaneous tissue, brain and eyes. |
| human infection with T. solium is by? | ingestion of raw undercooked meat. |
| the development of oncospheres into cysterci occurs in? | muscles of pigs/humans |
| the diagnostic stage of t. solium | egg/gravid proglottis |
| disease caused by T. Solium | cysticorcosis |
