General Pathology
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General Pathology - Marcador
General Pathology - Detalles
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Infections | Most common cause of inflammation |
Pain | Dolor |
Response of a living thing to trauma (inflammation) | Vital Reaction |
Mediators of inflammation | Humoral Response |
Movement of WBC's | Cellular Response |
Stacking of RBC's | Rouleaux formation |
Neutrophils | Polymorphonuclear cells |
Cells on the interior surface of blood vessel | Endothelial cells |
One of the 3 ways complement system can occur. Antibody/antigen complexes in an immune reaction | Classical pathway |
The common ending of the 3 pathways of complement system. Destroys cells drilling holes into plasma membrane. Act on leukocytes and endothelial cells. | Membrane attack complex |
Platelet aggregation, thrombosis | Thromboxane |
Opposes effects of thromboxane | Prostacyclin |
Smooth muscle contraction to slow blood flow down (so inflammatory reaction can take place) creates pain, fever. | Prostaglandins |
An immediate reaction to injury. Sudden onset, short duration. Typically mediated by neutrophils. | Acute inflammation |
Infection in bloodstream | Bacteremia |
Toxins in bloodstream | Toxemia |
Early stage of most inflammation. Viral infection. Joint swelling in rheumatoid arthritis. Mildest form of inflammation. Lesions heal without permanent consequences. | Serous Inflammation |
Caused by pus forming bacteria. | Purulent inflammation |
Form of ulcerative inflammation but has fibrino-purulent exudation. Includes mucus, cellular debris, fibrin, pus. | Pseudomembranous inflammation |
Thermorgulator | Endogenous pyrogens |
Non specific symptoms: fatigue, weakness, depression, lack of appetite, exhaustion. | Constitutional symptoms |